Crystal Structures and Nuclear Magnetic Resonance Studies of the Apo Form of the c-MYC:MAX bHLHZip Complex Reveal a Helical Basic Region in the Absence of DNA | Biochemistry
Genes | Free Full-Text | MYC Oncogene Contributions to Release of Cell Cycle Brakes
IJMS | Free Full-Text | Gene Transactivation and Transrepression in MYC-Driven Cancers
Structure and DNA sequence preferences of Myc and proteins that... | Download Scientific Diagram
Global mapping of c-Myc binding sites and target gene networks in human B cells | PNAS
Cell-type specific and combinatorial usage of diverse transcription factors revealed by genome-wide binding studies in multiple human cells. - Abstract - Europe PMC
MYC overexpression leads to increased chromatin interactions at superenhancers and c-Myc binding sites | bioRxiv
Dissecting transcriptional amplification by MYC | eLife
Sequence-specific DNA binding by MYC/MAX to low-affinity non-E-box motifs | PLOS ONE
Interaction of the oncoprotein transcription factor MYC with its chromatin cofactor WDR5 is essential for tumor maintenance | PNAS
footprintDB - a database of transcription factors with annotated cis elements (DNA motifs and sites) and binding interfaces
Dissecting transcriptional amplification by MYC | eLife
Frontiers | Strategies to target the cancer driver MYC in tumor cells
MYC: a complex problem: Trends in Cell Biology
Targeting MYC with modular synthetic transcriptional repressors derived from bHLH DNA-binding domains | Nature Biotechnology
Taking the Myc out of cancer: toward therapeutic strategies to directly inhibit c-Myc | Molecular Cancer | Full Text
The Disordered MAX N-terminus Modulates DNA Binding of the Transcription Factor MYC:MAX - ScienceDirect
Genes | Free Full-Text | MYC—Master Regulator of the Cancer Epigenome and Transcriptome